Due to the increasing prevalence of Alzheimer's Disease, these resources may
be of interest to some of you:
The Alzheimer's Association:
<http://www.alz.org/>
Recent research into the causes of Alzheimer's:
<http://www.alz.org/research/current/causes/1999/>
<http://www.alz.org/research/current/causes/2000/>
Recent research into the diagnosis and treatment of Alzheimer's:
<http://www.alz.org/research/current/diagnose/>
<http://www.alz.org/research/current/diagnose/2000/>
<http://www.alz.org/research/current/diagnose/1999/>
-------------------------------
Then, here is an extract from this weeks's latest news on Alzheiner's from
Nature Journal (2 Dec 2000):
A VACCINE FOR ALZHEIMER'S DISEASE?
<http://www.nature.com/nature/fow/001221.html>
Alzheimer's disease is characterised both by devastating dementia and
abnormal physical changes
in the brain. But it is not clear which, if any, of the pathological features
causes the dementia. Even
the prime suspects -- the plaques that form -- have not been definitely shown
to affect mental
function.
These plaques are build-ups of a peptide called amyloid-b which can
accumulate into dense protein
tangles. Although they are thought to be the most likely physical candidate
behind the mental
dysfunction, separating the plaques from mental function in order to test the
theory is very
difficult.
The door was opened last year by research showing that when amyloid-b was
used as a vaccine in
a mouse model of the disease, plaque formation was reduced (see Schenk et
al., below). Now,
two papers in this week's Nature make the link between plaque formation and
brain dysfunction,
providing the first real hope that it may be possible to ameliorate dementia
simply by disrupting the
deposition of amyloid-b. A third paper by Chen et al. adds validity to the
studies by
differentiating the effects of the disease from those of ordinary ageing in
mice.
For abstracts of all of the above mentioned articles, go to the Nature
website or to any of these other websites:
<http://www.sciencenews.org/sn_arc99/7_10_99/fob1.htm>
<http://www.looksmart.com/eus1/eus65300/eus65303/eus77824/eus541028/eus54733/e
us149342/eus93589/eus274205/eus71060/eus946526/r?l&> (Numerous Links on
Alzheimer's)
-----------------------------------------------
OTHER ORIGINS OF ALZHEIMER'S?
The following website discusses work done by certain scientists which
suggests that Alzheimer's and other modern scourges have their origins in
dubiously prepared or infected vaccines.
<http://www.inx.net/~carolynv/vaccines/nvic.htm>
Here are a few extracts from this site:
DISCOVERY OF AN ATYPICAL VIRUS INFECTING HUMANS LINKED TO VIRAL VACCINES
PRODUCED ON MONKEY TISSUES
In what could be one of the most important scientific discoveries of this
decade, an award winning pathologist and immunologist at the University of
Southern California, W. John Martin, M.D., Ph.D., has discovered an atypical
virus infecting both children and adults who are exhibiting neurological,
psychiatric and autoimmune disorder symptoms with diagnoses including chronic
fatigue syndrome, fibromyalgia, depression, schizophrenia, anxiety disorder,
seizures, developmental delays, autism, lupus, multiple sclerosis,
Alzheimer's, Parkinson's, unexplained encephalopathy and chronic vegetative
states.
Martin and his colleagues at USC's Infectious Diseases and Molecular
Pathology Laboratories have been meticulously culturing out stealth viruses
from patients for the past eight years and, in a stunning development earlier
this year, successfully identified one of the viruses as being of African
green monkey origin by using DNA sequence analysis. Kidney tissues from
African green monkeys have been used to make the live oral polio vaccine
(OPV) as well as other viral vaccines during the past three decades.
A distinctive feature of the virus Martin and his colleagues has
characterized is that it belongs to a novel class of atypical cytopathic
viruses (capable of causing pathologic changes in cells), which they refer to
as "stealth viruses" because they have the ability to evade detection by the
body's cellular defense mechanisms and appear to lack the antigens which
normally cause an inflammation typical of most infections that damage cells
and body tissues. The monkey-related stealth virus they are studying is a
cytomegalovirus belonging to the herpes virus family that causes an atypical
viral infection of the brain - a "stealth virus encephalopathy" - that can
produce a spectrum of disease symptoms without evoking an inflammatory
response.
Therefore, a person can also become infected and can carry and transmit the
virus to others without exhibiting symptoms. The stealth virus can remain
dormant in an infected but symptomless individual throughout life. However,
in some infected individuals the virus can become active, triggered perhaps
by significant mental or physical stress, and go on to cause atypical
responses to normal sensory input into the brain resulting in
sudden, unexplained neurological symptoms. It is thought that a stealth virus
can be transmitted, like HIV, hepatitis B or polio, by coming into direct
contact with the virus (such as ingesting or being injected with a
contaminated vaccine) or coming into contact with the blood or body fluids of
an infected individual.
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Dr Mel C Siff
Denver, USA
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http://www.egroups.com/group/supertraining
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