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It would be helpful to know who assayed the blood glucose using the PoCT
device, and whether it was a technique that allows visual reading as
well as the meter display. Was the anomalous PoCT result a single
occurrence, or repeatable?

Dr Jonathan Kay

On Monday, March 11, 2002, at 09:31 , Hogan, Tim wrote:

> Which system was used to measure a. The finger prick glucose, b. The
> urine
> ketones?
> Have seen no published evidence of hyperosmolar coma causing this
> problem
> since days of BM 1-44 stick. Has anyone else?
>
> Tim
> At Basildon
>
> [log in to unmask]
>
> Dear list members
>
> A young man presented with coma having been previously
> fit and well. The relatives gave a history of him
> having abdominal pain and vomiting over the last two
> days. At the A/E, he was found to be in hypovolaemic
> shock with peripheral shutdown. Despite this, a finger
> stick blood glucose was performed and the glucometer
> reading was 7.6 mmol/L. A urine test showed 4+ glucose
> but negative ketones. A venous blood sample was rushed
> to the lab which showed a serum glucose of 84.1 mmol/L
> and arterial blood gas sample showed severe metabolic
> acidosis with pH of 6.9. His serum B-hydroxybutyrate
> (BOHB)was > 12.0 mmol/L. The serum sodium was 152
> mmol/L, potassium 4.5 mmol/L, osmolality 430 mosm/L,
> urea 34 mmol/L and creatinine 481 umol/L. This young
> patient presented with unusual hyperosmolar DKA with
> predominant production of BOHB that gave a negative
> reaction with urine dip stick. Despite the glucometer
> SOP warning against use of finger stick sample in
> peripherally shut down patients, clinical staff seem
> to be unaware of this caveat.
>
> Comments are welcome.
>
> Mohammad
>
> =====
> Dr. M A Al-Jubouri
> Consultant Chemical Pathologist
>

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