At 00:33 31/03/99 +1000, you wrote:
>>In my opinion, this conception of selective "strengthening" of VMO is
>conceptually flawed. In patients with PFJ pain syndrome, it is not the
>strength, but activation pattern of the VMO/VL that is at fault. If the VMO
>is not being activated early enough during weightbearing flexion then the
>VL (larger xs area) will cause lateral maltracking. Standard rehab for this
>in Australia is to retrain selective timing/activation of VMO by using dual
>channel biofeedback in functional, weightbearing positions (eg lunge, 1/4
>squat, stepdowns), progressing to in-place plyometrics. Patellofemoral
>taping (a la McConnell) is vital if maltracking of glide or tilt variety is
>present.
>
>I cannot see how "strength" of the different components of the quads could
>be measured in isolation. Rather, EMG can be measured, which as we know
>does not measure force or torque but amount and timing of central
>activation of the muscle
>
>
>Matt McEwan
>B App Sc (Physio)
>M App Sc (Sports Physio)
>Sydney, Australia
>
>--------------------------------------------------------
However, in practical clinical applications for a great number of clients,
this seems to be a moot point. It is important for the clinician to know
what in physiological terms is being achieved. Most of my clients seem to
require a more straightforward explanation.
>
>
>
Owen Allen
Atherton Hospital
P.O. Box 183
Atherton 4883
Queensland, Australia.
Ph: 07 40910261
F: 07 40913502
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