Here are some references:

   Authors
    Weston P.  Lear J.
   Institution
    Leicester Royal Infirmary, UK.
   Title
    Gliomatosis cerebri or benign intracranial hypertension?.
   Source
    Postgraduate Medical Journal.  71(836):380-1, 1995 Jun.
   Abstract
    Benign intracranial hypertension is a condition of obscure aetiology
which
    presents with episodic headaches and nausea. It is a diagnosis based
on
    the exclusion of other intracranial pathology and computed tomography
is
    usually normal. We present a case of gliomatosis cerebri which was
    initially diagnosed and treated as benign intracranial hypertension.

   Authors
    Vassallo M.  Allen S.
   Institution
    Department of Medicine for the Elderly, Royal Bournemouth Hospital,
    Dorest, UK.
   Title
    An unusual cause of dementia.
   Source
    Postgraduate Medical Journal.  71(838):483-4, 1995 Aug.
   Abstract
    Gliomatosis cerebri is a rare cerebral tumour that presents with
    personality and mental state changes. Diagnosis can be very difficult
and
    many times is made at post mortem. We describe a 63-year-old man who
    presented initially with depression that merged into a
schizophrenia-like
    illness, and who developed progressive dementia prior to his death.
Two
    computed tomography (CT) scans of the brain were normal and the
diagnosis
    of gliomatosis cerebri was made at post mortem. The progressively
changing
    mental state was suggestive of an organic cause of his illness. Since
this
    tumour may not be detected by a CT scan, a magnetic resonance imaging
scan
    with T2-weighted images with spin echo sequences of the brain should
be
    performed. Prognosis is very poor but diagnosis is important to plan
    tumour may not be detected by a CT scan, a magnetic resonance imaging
scan
    with T2-weighted images with spin echo sequences of the brain should
be
    performed. Prognosis is very poor but diagnosis is important to plan
    terminal care. The patient described was unusual because he was older
than
    most people with this tumour, and he presented with psychiatric
symptoms
    which were thought to be non-organic for almost two years.

Authors
    Jennings MT.  Frenchman M.  Shehab T.  Johnson MD.  Creasy J.  LaPorte
K.
    Dettbarn WD.
   Institution
    Department of Neurology, Vanderbilt School of Medicine, Nashville,
USA.
   Title
    Gliomatosis cerebri presenting as intractable epilepsy during early
    childhood. [Review]
   Source
    Journal of Child Neurology.  10(1):37-45, 1995 Jan.
Abstract
    We review 160 cases of gliomatosis cerebri from the literature and
report
    an additional three infants and young children who presented with
    intractable epilepsy, corticospinal tract deficits, and developmental
    delay in whom a pathologic diagnosis was made. The progressive nature
of
    the encephalopathy in our cases was documented by serial clinical
   examination, electroencephalograms, magnetic resonance imaging, and
    positron emission tomographic scans. The natural history of
gliomatosis
    cerebri was determined by a retrospective review of the literature of
160
    cases in 85 reports. The most common neurologic symptoms and signs
    included corticospinal tract deficits (58%), dementia/mental
retardation
    (44%), headache (39%), seizures (38%), cranioneuropathies (37%),
increased
    intracranial pressure (34%), and spinocerebellar deficits (33%). The
most
    commonly involved central nervous system structures were the centrum
    semiovale and cerebrum (76%), mesencephalon (52%), pons (52%),
thalamus
    (43%), basal ganglia (34%), and the cerebellum (29%). Fifty-two
percent of
    patients were dead within 12 months of onset. Different grades of
glial
    neoplasm may also coexist within gliomatosis cerebri such as
astrocytoma
    with anaplastic astrocytoma, atypical or anaplastic oligodendroglioma,
and
    glioblastoma multiforme. Hypotheses regarding the pathogenesis of
    gliomatosis cerebri include blastomatous dysgenesis, diffuse
infiltration,
    gliomatosis cerebri include blastomatous dysgenesis, diffuse
infiltration,
    multicentric origin, in situ proliferation, and "field
transformation."
    The biologic determinants of whether a transformed glial cell behaves
as a
    relatively localized tumor mass or truly loses anchorage dependence to
    become migratory as well as proliferative are not understood.
[References:
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